Is Schistosoma japonicum cirrhosis serious?

Is Schistosoma japonicum cirrhosis serious?

Although cirrhosis of the liver can be said to be a common disease nowadays, people do not need to panic when they have cirrhosis of the liver. If it is in the early stage, if you pay attention to your diet and cooperate with the doctor's treatment, it can be completely cured. So do you know what schistosomiasis cirrhosis is? Is schistosomiasis cirrhosis serious?

Cirrhosis of the liver is caused by various causes, which lead to diffuse necrosis of hepatocytes, regeneration, hyperplasia of fibrous connective tissue, destruction of lobular structure, hyperplasia of pseudolobules and nodules. The main clinical manifestations are liver function damage and portal hypertension, and serious complications often occur in the late stage.

Medical history and symptoms: The causes of cirrhosis are diverse. In China, viral hepatitis, especially hepatitis B and C, is the main cause, followed by alcoholic hepatitis, schistosomiasis, etc. Because cirrhosis develops slowly or is in a long compensatory stage, the symptoms lack specificity, and liver function damage and portal hypertension symptoms only appear in the decompensated stage. Therefore, the patient's occupation and living habits should be understood in detail. The place of residence or history of contact with infected water. Conduct relevant examinations on patients suspected of hepatitis. Physical examination reveals dark complexion, spider angiomas, liver palms, and some patients may have jaundice, splenomegaly, ascites, open lateral circulation such as varicose veins of the abdominal wall and lower esophagus, gastric fundus, hemorrhoids, etc.

In compensated cirrhosis, liver function may be completely normal, and the diagnosis must be made in combination with medical history. Abnormal liver function in the degeneration stage, such as increased serum bilirubin, prolonged prothrombin time, increased ALT and AST to varying degrees, and increased AST/ALT ratio, reflects the severity of liver cell damage. GGT increases, especially in alcoholic cirrhosis, while serum albumin decreases and globulin increases, with the ratio between the two being inverted. Serum indicators of liver fibrosis include PⅢP and type Ⅳ collagen. In, FnR, HA, etc. increased.

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