What disease is pink foamy sputum

What disease is pink foamy sputum

Many people will catch a cold in winter due to the cold weather. A cold usually causes runny nose and cough. Even after the cold symptoms are gone, the cough will continue.

Therefore, you should take some cough medicine in time, otherwise it will cause tracheitis, tonsillitis and other diseases. If it is inflamed, there will be pink foam when spitting. So what disease is the pink foamy sputum?

Pink foamy sputum is caused by the infiltration of fluid from the capillaries into the pulmonary interstitium or alveoli. The most common clinical manifestation of pulmonary edema is acute heart failure. Because acute left heart failure causes increased pulmonary venous pressure, increased plasma exudation in the alveolar capillaries, and even capillary rupture, acute pulmonary edema is formed. This edema fluid contains protein and is serous or pink. Under the action of airflow, it will form foam, just like bubbles when the urine protein content increases.

Acute heart failure is often caused by certain triggers, which can cause patients with compensated heart function to suddenly develop heart failure, or cause patients who already have heart failure to suddenly worsen their condition. Therefore, it can occur in patients with normal cardiac function or without cardiac pathology.

Common causes include:

1. Acute diffuse myocardial damage: such as acute myocardial infarction, acute myocarditis, etc.

2. Acute cardiac afterload: such as a sudden significant increase in arterial pressure or hypertensive crisis; sudden excessive physical activity in patients with pre-existing valvular stenosis (mitral valve, aortic valve) or left ventricular outflow tract obstruction; rapid arrhythmia or excessive and rapid infusion.

3. Acute volume overload: Acute valvular reflux caused by acute myocardial infarction, infective endocarditis or trauma-induced papillary muscle rupture or dysfunction, chordae tendineae rupture, valvular perforation, etc., can lead to a significant increase in pulmonary venous pressure and acute pulmonary edema when excessive and rapid infusion occurs. After an acute left heart failure attack, if the primary cause and predisposing factors are eliminated, the patient can recover completely. If the patient survives and the underlying disease persists, chronic heart failure may develop. At this time, since various compensatory mechanisms have had sufficient time to work, the patient may have a period of stability, but heart failure may recur if there is a trigger again.

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