When a meningitis patient has chills, alternating hot and cold, restlessness, and difficulty controlling himself or herself, it may be a complication of purulent meningitis and needs to seek medical attention promptly and be treated by a professional doctor. Be sure not to adjust the condition in your own way. If coupled with a loss of appetite and general weakness and pain, the situation is more serious and requires timely medical attention. Once the diagnosis of purulent meningitis is confirmed, appropriate antibiotics should be used immediately for treatment. If the pathogen is clear, sensitive drugs should be selected against the pathogen. If the cause cannot be determined immediately, the drug can be selected according to the general pattern of disease. For example, for infections caused by meningococci and pneumococci, penicillin G2 may be the first choice; for infections caused by Haemophilus influenzae, ampicillin and tetracycline should be the first choice; for pneumococci, cephalosporins, chloramphenicol or canarin are the first choice; for Escherichia coli, ampicillin and cephalosporin are the first choice; for anaerobic bacteria and Proteus, kanamycin and gentamicin are the first choice; for Salmonella, ampicillin and chloramphenicol are the first choice; and for Pseudomonas aeruginosa, polymyxins and gentamicin are the first choice. If systemic administration is ineffective, it can be combined with intrathecal administration. If mixed infection with multiple pathogens is considered clinically, combination therapy is required. While using antibiotics, attention should also be paid to nutrition, water and electrolyte balance, prevention and treatment of cerebral edema and enhanced care. Administration of adrenocortical hormones while adequately using antibiotics can help control edema and reduce inflammatory responses. Common complications of purulent meningitis include intracranial purulent infectious diseases such as subdural effusion, pus, brain abscess, cerebral infarction, venous sinus thrombosis, and extracranial lesions such as bacterial endocarditis, pneumonia, purulent arthritis, nephritis, cyclitis, and even disseminated intravascular coagulation. Sequelae include phlegm disease, hydrocephalus, aphasia, limb paralysis and cranial nerve palsy. |
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