Genetic yellow skin

Although we are all yellow people, our skin is not that yellow, just a little bit. Some people have jaundice after birth, so the skin on their body turns yellow. It can be cured slowly with recovery. For hereditary yellow skin, this situation still depends on the individual's pathological condition, because after all, everyone's physical condition is different.

Symptoms and signs

Hereditary skin diseases are caused by parents passing their abnormal or mutated genes to the next generation, causing the next generation to develop skin lesions specific to the gene at birth or later. There are very few non-hereditary congenital skin diseases, which are mainly diseases of the skin and its appendages caused by the interference of nutrition, viruses or microbial infections, or chemical and physical factors during the development of the fetus in the mother's body.

①The general characteristics of hereditary skin diseases are the same as those of hereditary diseases;

②It can be present at birth or appear in childhood or adolescence;

③The symptoms are not necessarily unique to this hereditary skin disease. For example, the blisters caused by friction are similar to those of congenital epidermolysis bullosa.

④ The same disease may manifest in different people with different symptoms. For example, mild ichthyosis may only cause slight changes in the limbs, while severe ichthyosis may affect the trunk and limbs, with prominent keratinization and dryness, and may also be accompanied by follicular keratinization and palmoplantar keratinization.

⑤ The same disease may have different inheritance patterns. For example, ichthyosis vulgaris is inherited in an autosomal dominant manner, while another type of the disease is inherited in a sex-linked recessive manner.

Common hereditary skin diseases: There are more than a hundred known hereditary skin diseases, such as freckles, ichthyosis, albinism, etc.

Medication

Most of them have no effective treatment and cannot be eradicated. Some diseases can be improved through medication or diet therapy. For example, hereditary angioedema is caused by autosomal dominant C1 esterase inhibitor deficiency, which can be treated with kallikrein and the androgen-like drug danazol. Phenylketonuria can be prevented by restricting dietary phenylalanine intake. Acrodermatitis enteropathica can be controlled by zinc preparations. Some congenital enzyme defects or immunoglobulin deficiency can be treated by supplementing the missing substances. But these treatments do not remove the cause of the disease. When some congenital malformations or abnormalities affect function or beauty, surgical treatment can be adopted according to the specific circumstances.