What is the best way to treat corneal leukoplakia

What is the best way to treat corneal leukoplakia

The cornea is the most important part of the eye. Human vision can only reach the retinal nerves through filtering through the cornea, and the cornea is often a high-incidence site for lesions. For example, corneal leukoma is a disease that is more prone to sudden onset. Unlike cataracts, corneal leukoma is more likely to occur in young and middle-aged people. Let's take a look at the final introduction on how to treat corneal leukoma.

1. Introduction: Corneal leukoplakia is the most common eye disease in ophthalmology. It can be caused by infection or trauma. It not only affects the appearance but also the visual function. It is especially harmful to the physical and mental health of young people. If it is not treated in time, more and more new blood vessels will form on the leukoplakia, which will affect the efficacy of later treatment.

2. Pathogenesis: Fungal leukoma is an infectious corneal disease caused by pathogenic fungi and has a very high blindness rate. Fungal keratitis has a slow onset and a long course, which can last up to 2 to 3 months, and corneal ulcers often appear within a few days of onset. The onset is slow, the course is subacute, the irritation symptoms are mild, and there is visual impairment. The corneal infiltrates are white or gray, dense, dull in surface, and have a toothpaste-like or moss-like appearance.

3. Treatment method: Please shake well before use. The best starting dose for the treatment of fungal keratitis with 5% natamycin is 1 drop each time, once every 1-2 hours, dripped into the conjunctival sac. After 3-4 days, change to 1 drop each time, 6-8 times a day. Treatment usually lasts for 14-21 days, or until active fungal keratitis subsides. If the disease can be diagnosed early and given reasonable treatment early, satisfactory results can be achieved for mild and moderate cases. However, due to misdiagnosis and incorrect treatment, the opportunity is missed, leading to corneal stromal necrosis, anterior chamber abscess, endophthalmitis and glaucoma, and in severe cases, loss of the eyeball.

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