Cerebral infarction is generally caused by lack of oxygen in the brain. It is difficult to completely cure this disease, mainly because this disease will leave many sequelae after it occurs. These sequelae will more or less affect the patient's future life and physical condition. Therefore, you should try your best to control the development of your disease and avoid complications. Pay more attention to rest during the treatment process so that your brain can be fully maintained. If the condition is relatively serious, it is best to rest in bed, especially if convulsions occur, you must pay special attention to it and take medication to relieve it. treat 1. General treatment in the acute phase During the acute phase, patients should rest in bed as much as possible and strengthen the care of the skin, oral cavity, respiratory tract, bowel and urination. Pay attention to the balance of water and electrolytes. If patients are still unable to eat on their own 48 to 72 hours after onset, they should be given nasogastric liquid diet to ensure nutritional supply. The patient's daily care, diet, and treatment of other complications should be given priority. In addition, most patients, their relatives and some medical staff hope that there will be better medicines to help patients recover soon, but ignore other aspects of treatment, such as the patient's diet. Since some patients with cerebral infarction cannot take care of themselves and even have difficulty swallowing during the acute phase, problems with their metabolism will quickly arise if they are not given proper nutrition and energy. At this time, even the best treatment medications will hardly produce good results. 2. Treatment of cerebral edema (1) Mannitol: 20% mannitol hypertonic solution is commonly used in clinical practice. Mannitol is one of the most commonly used and effective dehydrating agents. (2) 10% glycerol fructose can produce pharmacological effects through hypertonic dehydration. It can also utilize the energy generated by glycerol metabolism into the brain metabolism process, thereby improving local metabolism. Through the above effects, it can reduce intracranial pressure and intraocular pressure, eliminate cerebral edema, increase cerebral blood volume and cerebral oxygen consumption, and improve brain metabolism. (3) Diuretic dehydration agents such as furosemide (Lasix) and sodium urate can be injected intermittently intramuscularly or intravenously. |
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